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Given that KIR expression is fixed during NK cell development, this data suggests 3 KIR/HLA: Genetic Clues for a Role of NK Cells in the Control of HIV 33 that these changes are likely related to the preferential expansion of KIR + NK cells with disease progression HIV infection. Furthermore, KIR3DS1+ NK cells exhibit enhanced function during early infection [56] and have been shown to expand preferentially following acute HIV infection in subjects that co-express its putative ligand, HLA-Bw480I [57].

Activating KIR with short intracellular tails lack these inhibitory motifs but contain a charged lysine residue in their transmembrane domain that allows interaction with ITAM-containing adaptor proteins that transmit the activating signal to NK cells [32] following ligation [33]. The known ligands of the KIR receptors consist of MHC-class I receptors and homologues. Several groups have speculated that rapid evolution at the MHC locus may have driven the co-evolution of their binding KIRs in response to infections in higher mammals [31].

Vasudevan S, Tong Y, Steitz JA (2007) Switching from repression to activation: microRNAs can up-regulate translation. Vigorito E, Perks KL, Abreu-Goodger C, Bunting S, Xiang Z, Kohlhaas S, Das PP, Miska EA, Rodriguez A, Bradley A, Smith KG, Rada C, Enright AJ, Toellner KM, Maclennan IC, Turner M (2007) MicroRNA-155 regulates the generation of immunoglobulin class-switched plasma cells. T. D. Katsikis 63. Wu H, Neilson JR, Kumar P, Manocha M, Shankar P, Sharp PA, Manjunath N (2007) miRNA profiling of naive, effector and memory CD8 T cells.

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a practical approach to identifying and tracking unauthorized 802.11 cards and access points


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